That’s odd to hear. I take 5 HTP every day. Up to 200 mg. It’s tryptophan(sp) serotonin precursor.
Makes me feel great. I try to only take it as needed.
Do you try and be natural for the most part?
Try too. Almost there if it weren’t for nicotine.
I give you some serious street cred. I tried being without an AP, just taking an antidepressant and klonopin. That was hell, even worse when I didn’t have kpins. I was going to the liquor store every night. We are all different, just maybe you will overcome this with your own yoda training. 
We’ll see how it plays out. I live a very stress free life. Especially these last couple weeks where I’ve learned to just stay at home.
Had the census bureau stop by. It was right after I went to the grocery store and was sort of wound up. Had to do an interview to help them with there stats.
Aside from that I haven’t done anything or seen anyone all day. Practically no triggers aside from the internal telepathy thing when I think about someone when I’m thinking something. Or some ■■■■ haven’t really figured that one out. When I forget about it though it seems to go away.
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I think my mind just might be tired of words in general.
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I unfortunately need to be in public, or I will go ape ■■■■ in my house. I have days where I don’t leave and sleep all day, but still get the fan in my room, voices talking to me. Yea if I don’t think about them. They stop. But I’m so use to listening for voices that my brain just does it. 
working actually isn’t too bad for me, it keeps my mind off my problems. Most the time, or I stand there bored. Thinking about my problems and how my back hurts and that I’m going to be physically handicap someday…just doom, is all I think about.
Ah you gotta relax man. Build up some resistance to the thinking you don’t like. I’ve been doing great all day but I’ve been awake since 5:30 and I’m getting kind of sloppy and careless. Still the best day I’ve had so far. A sign of things to come.
And I’m kind of dependent on following along with this site to keep my thinking afloat. My mind refuses to be entertained by itself.
Yea it keeps me out of my head. I was going to go to AA tonight but got late.
Just a couple more hours and then I can crash.
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It’s hard to understand your English. I’m guessing English isn’t your first language and that you’re using a translator? I’m interested in what you have to say but it’s kind of hard to follow. Thanks for posting this stuff.
Said he was using a translator.
as I said (in this or in another post), the problem of serotonin would benefit from a dedicated discussion. I was referring to SSRIs however, not the tryptophan (however schizophrenics must also stay away from tryptophan). And I repeat: I am not anti-medicine, I do not say “no SSRIs, tryptophan yes” without reason. I can not explain the reason well, but at the biological level, block the serotonin transporter has a completely different effect from taking tryptophan, the effect it has on receptors is different. In fact, knowing the effect of blocking the serotonin transporter, before reading the research regarding the intake of tryptophan and about the effect of tryptophan hidroxylase (actually not entirely clear because little studied), I was convinced that hiring tryptophan could do badly as to block the serotonin transporter, but in fact the studies seem not confirm my assumption, it seems that there may be beneficial effects obtained by taking tryptophan … not all the research pointing in same direction, and there are still side effects (such as tinnitus sufferers noon should hire him, and perhaps even those suffering from schizophrenia), but generally do not consider myself a practicing “evil” … taking SSRIs, however, is how much more you can do wrong! If you seek 5HTTLPR laws and some research, you’ll make us realize (block SERT causes depression), but there are many other scientific evidence that block the serotonin transporter HURTS. The only reason to take an SSRI is not as devastating as expected, is because even the most selective of these drugs it is not very selective, and this mask the negative effects of the blockade of the serotonin transporter … this looks good, but it is not: if they were really selective on SERT, would be even more obvious that the use of these drugs is wrong (in fact it is already clear that, just read the research, which doctors do not do), because the Studies conducted by the pharmaceutical companies would be disastrous; But now studies of pharmaceutical companies are on the cutting edge, hiding those that they do not agreed http://www.spring.org.uk/2008/02/new-study-ssri-antidepressants-dont.php
but this only because in addition to blocking the serotonin transporter, these drugs are also other, if only block the serotonin transporter, there would be more suicide. but their other effects ( positive ) , mask those of serotonin . But the budget is not positive .
Who exactly are they studying?
I have done research studies for the betterment of patients with schizophrenia, including blood draws and heavy questionnaire, in which I got paid nearly $500 over 8 weeks time.
Everyone talks about the mystery of this illness, and if you are saying doctors don’t know their stuff, I would have to differ. We can try to do well in lifestyle and health, but we need our docs.
I’m sorry, I can not make myself understood.
All the talk about the futility of SSRIs and ignorance of doctors, was related to the treatment of depression and to it alone, not schizophrenia. Regarding the treatment of depression I take full responsibility, when I say that the scientific evidence goes against the practice of medicine and that doctors are ignorant of this aspect, and I can prove it. I do not gain anything, to convince you, but for intellectual honesty, if I am sure of one thing, I have to say it, respecting the views of others. I’m not saying you have to do without the doctors, I am a person of science and are in favor of the medicine, the problem is that even the medicine makes mistakes. There are many drugs that can be used for depression that do not block the serotonin transporter and that should be used instead of SSRIs, but they have to be doctors to understand, and I hope that they will soon, just read the studies.
Regarding the treatment of schizophrenia I’m not an expert, and you’re right: among all the noise is the least understood and most difficult to understand. And mind you: I AM NOT AGAINST THE DOCTORS. As for the relationship between schizophrenia, serotonin and tryptophan, there are some points that seem clear enough:
- Inflammatory processes alter the metabolism of tryptophan and block these processes decreases the symptoms of schizophrenia
- Starting on tryptophan metabolism are synthesized substances strongly implicated in schizophrenia
- Schizophrenia has a very close relationship with certain serotonin receptors, particularly the 5HT2a receptor (on this point should focus much doctors, because that kind of receptor can be modulated in different ways depending on the substance used)
- Block certain serotonin receptors, attenuates the negative symptoms
In addition there is no evidence that using SSRIs helpful in schizophrenia, and this was easily predictable, there is no evidence that can suggest a use of SSRIs. For web surfing in the use of supplements of tryptophan (I should mention, has a different effect than the SSRIs), one could hypothesize that may be of some use, but studies have not confirmed this hypothesis and, indeed, warn which can be counter-productive (read here http://www.ncbi.nlm.nih.gov/pubmed/369623). This does not mean you have to limit the tryptophan from the diet (which brings other problems).
So a person responsible and wise, would say that it is better not to take supplements of tryptophan in the case of schizophrenia, and it is better not to eliminate from the diet tryptophan: simply need to keep the normal intake.
The following studies on schizophrenia
PS: If you want me to deepen the discussion about SSRIs vs depression I have to open a separate discussion, but the problem of these discussions is that, although I do see that what doctors do, is wrong, patients have no choice but to relying on doctors (and rightly so). I would not convince patients, I would that doctors would take awareness of their mistakes to improve the treatment of their patients.
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I hope I have made it clear that I do not invite anyone to do without their doctors, that’s not my purpose.
Continuing the discussion from Hi to all!:
to be precise are not against the use of nootropics. I am against the use of racetam. The only racetam would try (but need a prescription), it is levetiracetam. Someone would ask, but how would you use not piracetam that is quieter and instead levetiracetam you? I think that even if the levetiracetam seems to have more side effects, is the most studied, has been used for many years by many more people at doses much higher than I would use. Levetiracetam increases slow wave sleep (sleep nootropic is the number 1 to be considered, the number 2 is aerobic exercise), also recent studies are promising in the treatment of Alzheimer’s.
Alphabrain contains Huperzine-A, which increases acetylcholine, it is an excellent nootropic but can cause melancholy and lead to depression in those who are at risk, but certainly enhances memory. before taking something so strong I would start taking more choline . It is simpler , safer, and leads to some results start using a precursor , rather than immediately taking a substance that has an effect so ’ strong . Also you have to consider this: studies on smokers show that when you stop the intake of nicotine on the brain is not plastic ; This is obvious because stopping to take a substance descend to a level lower than that of departure. So when you start using a substance like that , or you know for sure how long I can take it while maintaining tolerance to a minimum (basically doing cycles ) , or it takes forever Alpha-GPC is a precursor of acetylcholine, but I think that the nootropic effect of this substance is not due only to the increase of acetylcholine. Bacopa Monniera… i not would hire him as first choice because I do not know very well how it works. It sounds interesting for the effect on memory Chronic Effects of Brahmi (Bacopa monnieri) on Human Memory | Neuropsychopharmacology. I read a study where Bacopa increases the activity of tryptophan hydroxylase and this intrigues me, because unlike the increase of serotonin given by blocking the transporter, which predisposes to emotional disorders, could have the opposite effect, and this would make it a good nootropic, but unfortunately there are few studies.
I prefer to start with nootropics who also are clearly anxiolytic and antidepressant
A great base is:
sarcosine. + Uridine. + Theanine. + N-acetylcysteine. + Omega-3. + Alpha-gpc
After I could also consider the use of Huperzine and Bacopa (which I guess have better effects), but very carefully. Anyway consider alpha brain much better than racetam, that’s for sure. I also think it might be a good help for certain types of ADHD. Some studies report that different types of ADHD are associated with different genetic traits, one example is associated with a low intake of choline … as I said the nootropic effect of choline think is also given to other factors other than its conversion acetylcholine, so it’s a good start taking a supplement of choline, but for sure if a person has a defect in absorption of choline, the benefits resulting structure also using the Huperzine-A. I never tire of saying, however, that we must be careful with these things, improve one thing it may worsen another. For example ADHD also has positive aspects, creativity, extraversion …
There are also a lot of other substances that are very interesting from the point of view nootropic (requiring a prescription or otherwise).