This post should be twinned with:
As it talks about the same mechanism of mitochiondrial targeting, but this post is an actual drug doing that! 
Are you sure? I didn’t see any mention of mitochondria in this article.
“There’s a particular type of neuron, cells called parvalbumin (PV) cells, that inhibits the activity of other neurons in the brain”
Which is mitochondrial targeting.
From what I’m reading parvalbumin cells and mitochondria have a very complex relationship. I’m not trying to be a dick but both pieces of research were also published by two different labs. I’m pretty foggy this afternoon so it’s totally possible I’m missing something but right now I don’t see the connection.
I don’t claim to know anything about it.
I just extrapolated this quote “The Genomic Press Interview highlights Do’s current research focus on parvalbumin interneurons (PVI), which are crucial for cognitive, affective, and social activities and are impaired in patients with schizophrenia. Her team’s work suggests that mitochondria-targeted antioxidants could significantly alleviate symptoms and improve cognitive functions in schizophrenia patients”
And put two and two together.
I don’t know what I am talking about really…
I see what you’re saying. This article is also super vague. I assumed “relaxing excited neurons” was referring to glutamate in some way.
my antipsycotic lurasidone target parvalbumin neurons and have restorative effect on them.