This woman was part of the stem cell therapy research with Dr Lodge. The article above is not written by someone who understands the research - I’m quite sure the scientists involved never said anything about delusions in rodents.
Fortunately the actual paper is public. In it she describes the therapy and how they used it to identify the brain circuits that could be modulated to normalize dopamine activity (positive symptoms) and how their therapy also fixed one aspect of cognitive dysfunction, extradimensional set-shifting, but not reversal learning.
Unlike reversal learning, we found that gene therapy to over-express the α5 subunit of the GABAA receptor improves schizophrenia-like deficits in extradimensional set-shifting as measured by the attentional set-shifting test…
This deficit was completely abolished in the animals that received gene therapy to over-express the α5 subunit in pyramidal cells of the vHipp.
This experiment did not address negative symptoms and the researcher seems to think they have a different neural origin.
In the current experiments, we focused on pathways from the vHipp to the NAc and mPFC. However, schizophrenia is a heterogeneous disorder and the vHipp is not the only site of pathology. For example, structural and functional changes have also been observed in the thalamus of schizophrenia patients59 and we have recently demonstrated that the paraventricular nucleus of the thalamus can also regulate dopamine signaling via the NAc60. Further, the vHipp sends and receives many projections to and from other brain regions beyond the NAc and mPFC. For example, reciprocal connections exist between the basolateral amygdala (BLA) and vHipp61 and optogenetic inhibition of this BLA-vHipp pathway has been shown to decrease anxiety-like behavior62 and increase social interaction time63, which has been used to model negative symptoms of the disorder. Conversely, activation of the pathway increases anxiety-like behaviors62 and decreases social interaction time63. The BLA has been implicated in schizophrenia64, therefore, it is likely that changes in this neural circuit may also contribute to the pathology of schizophrenia.